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Old 06-09-2006, 02:14 PM
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Default Trenbolone breathlessness

Anyone ever experience breathlessness while on Trenbolone? I've heard it's quite common.

After about my 3rd inject of every other day 100mg I think it hit me. I'm not sure if I'm imagining it. I breath ok but it seems to get hard to draw a deep breath. When I yawn or breath deep it's like I can't complete it.

I thought if someone else has experienced you might put my mind at ease. I stopped it and in a day I was ok. Am I imagining it or is this typical?
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Old 06-09-2006, 02:30 PM
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I have heard of "Tren Cough" before, but I was under the impression that it was a result of accidentally injecting Tren in to a vein...Hmmmm...
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Old 06-09-2006, 07:42 PM
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Quote:
Originally Posted by TRAZ
I have heard of "Tren Cough" before, but I was under the impression that it was a result of accidentally injecting Tren in to a vein...Hmmmm...
This is not true. Let me try to find the article that explains it...
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Old 06-09-2006, 07:43 PM
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Tren- Fat burning and "fina cough" both from prostaglandin metabolization Posted on AR & IBB as well-


It's been widely disussed of Trens fat burning properties through rises in IGF and Prostaglandins. While IGF is a fairly well known substance in the bodybuilding world today, prostaglandins are fairly unknown in terms of formation and roles in the body.
So below, a brief dicription of prostoglandins and their role in fat burning, "fina cough", and why a person going through Tren administration can experience it's fat burning effects without the dreaded "Cough"

The term prostaglandin comes from the word-Prostate. The first prostoglandins were first dicovered in semen about the mid 1930's and it was thought that prostaglandins were made from the prostate. Since this time, it has been dicovered that most prostaglandins are not even constructed in the prostate.

Prostaglandins are made by two different pathways(Cyclooxygenase and Lipoxygenase), and considering prostaglandins are a group of about 20 lipid cells, they have contrary function; responsible for stimulating as well as alleviating inflammation(Inflammation stimulation is the rapid metabolism of them expelled through the bronchials), regulate blood flow to particular organs, control ion transport across membranes, modulate synaptic transmission, induce sleep, mediate lipid release, and regulate metabolism is various tissue.

Prostaglandins are synthesized from arachidonate(Lipoxygenase which catalyze the dioxygenation of polyunsaturated fatty acids) in the cell membrane by the action of phospholipase A2. Cyclooxygenase and lipoxygenase pathways, compete with one another to form prostaglandins(as well as thromboxane or leukotriene-leukotriene being a bronchial stimulator),
In the cyclooxygenase pathway, the prostaglandins D, E and F plus thromboxane and prostacyclin are made. Thromboxanes are made in platelets and cause constriction of vascular smooth muscle and platelet aggregation
Leukotrienes are made in leukocytes and macrophages via the lipoxygenase pathway. They are potent constrictors of the bronchial airways. They are also important in inflammation and hypersensitivity reactions as they increase vascular permeability.

Being that prostaglandins from either pathway, are still fatty acids of a group, they mediate lipid release and controll tissue metabolization, so fat burning is a luxerry of either pathway of formation. It's the pathway from which they are constructed that dictates "fina cough". As prostaglandins made from the Cyclooxygenase pathway dictate muscle constriction and platlet aggregation, and the Lipoxygenase pathway dictates bronchial constriction(the main form of expulsion)


Refs:
Cackatoo Press
Columbia Encyclopedia 6th Edition
Science Daily Magazine
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Old 06-09-2006, 11:17 PM
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I'm not too fond of tren. Some people like it ...I don't
I just think that in my opinion has too many sides to mess with especially for just f*cking around in the gym for fun. Just my opinion
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Old 06-09-2006, 11:22 PM
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at 100mg ED i was short of breath constantly, it scared me so i stopped taking it. it wasnt jsut after shots it was all day...
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Old 06-10-2006, 07:45 AM
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Quote:
Originally Posted by rckpytn
at 100mg ED i was short of breath constantly, it scared me so i stopped taking it. it wasnt jsut after shots it was all day...
Me, too. At 100mg is when it hit.

It's not like a gasping for breath, I can breath fine shallowly, but I can't seem to take a full satisfying deep breath, it's like I can't complete it. Is that your experience?
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Old 06-10-2006, 07:48 AM
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Quote:
Originally Posted by tribal
Tren- Fat burning and "fina cough" both from prostaglandin metabolization Posted on AR & IBB as well-


It's been widely disussed of Trens fat burning properties through rises in IGF and Prostaglandins. While IGF is a fairly well known substance in the bodybuilding world today, prostaglandins are fairly unknown in terms of formation and roles in the body.
So below, a brief dicription of prostoglandins and their role in fat burning, "fina cough", and why a person going through Tren administration can experience it's fat burning effects without the dreaded "Cough"

The term prostaglandin comes from the word-Prostate. The first prostoglandins were first dicovered in semen about the mid 1930's and it was thought that prostaglandins were made from the prostate. Since this time, it has been dicovered that most prostaglandins are not even constructed in the prostate.

Prostaglandins are made by two different pathways(Cyclooxygenase and Lipoxygenase), and considering prostaglandins are a group of about 20 lipid cells, they have contrary function; responsible for stimulating as well as alleviating inflammation(Inflammation stimulation is the rapid metabolism of them expelled through the bronchials), regulate blood flow to particular organs, control ion transport across membranes, modulate synaptic transmission, induce sleep, mediate lipid release, and regulate metabolism is various tissue.

Prostaglandins are synthesized from arachidonate(Lipoxygenase which catalyze the dioxygenation of polyunsaturated fatty acids) in the cell membrane by the action of phospholipase A2. Cyclooxygenase and lipoxygenase pathways, compete with one another to form prostaglandins(as well as thromboxane or leukotriene-leukotriene being a bronchial stimulator),
In the cyclooxygenase pathway, the prostaglandins D, E and F plus thromboxane and prostacyclin are made. Thromboxanes are made in platelets and cause constriction of vascular smooth muscle and platelet aggregation
Leukotrienes are made in leukocytes and macrophages via the lipoxygenase pathway. They are potent constrictors of the bronchial airways. They are also important in inflammation and hypersensitivity reactions as they increase vascular permeability.

Being that prostaglandins from either pathway, are still fatty acids of a group, they mediate lipid release and controll tissue metabolization, so fat burning is a luxerry of either pathway of formation. It's the pathway from which they are constructed that dictates "fina cough". As prostaglandins made from the Cyclooxygenase pathway dictate muscle constriction and platlet aggregation, and the Lipoxygenase pathway dictates bronchial constriction(the main form of expulsion)


Refs:
Cackatoo Press
Columbia Encyclopedia 6th Edition
Science Daily Magazine
Thanks, good article. Is there any way to counteract it though? What about small doses of clen to counteract it as a bronchodilator?
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